Impact of pnpR, a LysR-type regulator-encoding gene, on the cellular processes of Pseudomonas putida DLL-E4

Qiongzhen Chen, Hui Tu, Fei Huang, Yicheng Wang, Weiliang Dong, Wenhui Wang, Zhoukun Li, Fei Wang, Zhongli Cui

Research output: Contribution to journalArticlepeer-review

6 Scopus citations

Abstract

LysR-type transcriptional regulators (LTTRs) regulate various cellular processes in bacteria. pnpR is an LTTR-encoding gene involved in the regulation of hydroquinone (HQ) degradation, and its effects on the cellular processes of Pseudomonas putida DLL-E4 were investigated at the physiological, biochemical and molecular levels. Reverse transcription polymerase chain reaction revealed that pnpR positively regulated its own expression and that of the pnpC1C2DECX1X2 operon; additionally, pnpR partially regulated the expression of pnpA when P. putida was grown on para-nitrophenol (PNP) or HQ. Strains DLL-E4 and DLL-ΔpnpR exhibited similar cellular morphologies and growth rates. Transcriptome analysis revealed that pnpR regulated the expression of genes in addition to those involved in PNP degradation. A total of 20 genes were upregulated and 19 genes were downregulated by at least 2-fold in strain DLL-ΔpnpR relative to strain DLL-E4. Bioinformatic analysis revealed putative PnpR-binding sites located in the upstream regions of genes involved in PNP degradation, carbon catabolite repression and other cellular processes. The utilization of L-aspartic acid, L-histidine, L-pyroglutamic acid, L-serine, ? -aminobutyric acid, D,L-lactic acid, D-saccharic acid, succinic acid and L-alaninamide was increased at least 1.3-fold in strain DLL-ΔpnpR as shown by BIOLOG assays, indicating that pnpR plays a potential negative regulation role in the utilization of carbon sources.

Original languageEnglish
Article numberfnw110
Pages (from-to)1-10
Number of pages10
JournalFEMS Microbiology Letters
Volume363
Issue number12
DOIs
StatePublished - 1 Jun 2016

Keywords

  • Carbon source utilization
  • Cellular process
  • Gene regulation
  • PNP degradation
  • PnpR deletion
  • Pseudomonas putida DLL-E4

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